1. Vitamin D and intestinal calcium absorption / S. Christakos [et al.] // Mol. Cell Endocrinol. - 2011. - Vol. 347, N 1-2. - P. 25-29. https://doi.org/10.1016/j.mce.2011.05.038
2. Calcitriol controls the epithelial calcium channel in kidney / J. G. Hoenderop [et al.] // J. Am. Soc. Nephrol. - 2001. - Vol. 12, N 7. - P. 1342-1349.
3. Structural organization of the human vitamin D receptor chromosomal gene and its promoter / K.-I. Myamoto [et al.] // Mol. Endocrinol. - 1997. - Vol. 11, N 8. - P. 1165-1179. https://doi.org/10.1210/me.11.8.1165
4. The caudal-related homeodomain protein Cdx-2 regulates vitamin D receptor gene expression in the small intestine / H. Yamamoto [et al.] // J. Bone Miner. Res. - 1999. - Vol. 14, N 2. - P. 240-247. https://doi.org/10.1359/jbmr.1999.14.2.240
5. The polymorphism in the caudal-related homeodomain protein Cdx-2 binding element in the human vitamin D receptor gene / H. Arai [et al.] // J. Bone Miner. Res. - 2001. - Vol. 16, N 7. - P. 1256-1264. https://doi.org/10.1359/jbmr.2001.16.7.1256
6. Cdx-2 polymorphism in the promoter region of the human vitamin D receptor gene determines susceptibility to fracture in the elderly / Y. Fang [et al.] // J. Bone Miner. Res. - 2003. - Vol. 18, N 9. - P. 1632-1641. https://doi.org/10.1359/jbmr.2003.18.9.1632
7. The vitamin D receptor gene start codon polymorphism: a functional analysis of FokI variants / C. Gross [et al.] // J. Bone Miner. Res. - 1998. - Vol. 13, N 11. - P. 1691-1699. https://doi.org/10.1359/jbmr.1998.13.11.1691
8. Zmuda, J. M. Molecular epidemiology of vitamin D receptor gene variants / J. M. Zmuda, J. A. Cauley, R. E. Ferrell // Epidemiol. Rev. - 2000. - Vol. 22, N 2. - P. 203-217. https://doi.org/10.1093/oxfordjournals.epirev.a018033
9. Lips, P. Vitamin D defciency and secondary hyperparathyroidism in the elderly: consequences for bone loss and fractures and therapeutic implications / P. Lips // Endocr. Rev. - 2001. - Vol. 22, N 4. - P. 477-501. https://doi.org/10.1210/er.22.4.477
10. Vitamin D receptor gene polymorphisms are associated with the risk of fractures in postmenopausal women, independently of bone mineral density. The OFELY study / P. Garnero [et al.] // J. Clin. Endocrinol. Metab. - 2005. - Vol. 90, N 8. - P. 4829-4835. https://doi.org/10.1210/jc.2005-0364
11. Molecular nature of the vitamin D receptor and its role in regulation of gene expression / P. W. Jurutka [et al.] // Rev. Endocr. Metab. Disord. - 2001. - Vol. 2, N 2. - P. 203-216.
12. Association between vitamin D receptor gene polymorphisms and bone mineral density in Chinese women / Y. Li [et al.] // Mol. Biol. Rep. - 2012. - Vol. 39, N 5. - P. 5709-5717. https://doi.org/10.1007/s11033-011-1380-3
13. Association between vitamin D receptor gene BsmI polymorphism and bone mineral density in a population of 146 Iranian women / F. Pouresmaeili [et al.] // Cell J. - 2013. - Vol. 15, N 1. - P. 75-82.
14. Relation of BsmI vitamin D receptor gene polymorphism to bone mineral density and occurrence of osteoporosis in postmenopausal Chinese women in Taiwan / H.-Y. Chen [et al.] // Osteoporosis Int. - 2001. - Vol. 12, N 12. - P. 1036-1041. https://doi.org/10.1007/s001980170014
15. Майлян, Э. А. Влияние полиморфизма 283 A>G (BSMI) гена рецептора витамина D на развитие остеопороза у женщин в постменопаузе / Э. А. Майлян // Мед. вестн. Юга России. - 2016. - № 4. - С. 32-38.
16. Association between polymorphisms of VDR, COL1A1, and LCT genes and bone mineral density in Belarusian women with severe postmenopausal osteoporosis / P. Marozik [et al.] // Medicina (Kaunas). - 2013. - Vol. 49, N 4. - P. 177-184.
17. Vitamin D receptor BsmI polymorphism and osteoporosis risk: a meta-analysis from 26 studies / Fu Jia [et al.] // Gen. Test. Mol. Biomarkers. - 2012. - Vol. 17, N 1. - P. 30-34. https://doi.org/10.1089/gtmb.2012.0267
18. Reference SNP (rs) Report: rs7975232 // National Center for Biotechnology Information [Electronic resource]. - Mode of access : https://www.ncbi.nlm.nih.gov/projects/SNP/snp_ref.cgi?rs=7975232. - Date of access : 07.04.2019.
19. Vitamin D receptor polymorphism, bone mineral density, and osteoporotic vertebral fracture: studies in a UK population / L. A. Houston [et al.] // Bone. - 1996. - Vol. 18, N 3. - P. 249-252. https://doi.org/10.1016/8756-3282(95)00483-1
20. Polymorphism of the gene for vitamin D receptor, bone mass, and bone turnover in women with postmenopausal osteoporosis / R. Fontova Garrofé [et al.] // Rev. Clin. Esp. - 2000. - Vol. 200, N 4. - P. 198-202. https://doi.org/10.1016/S0014-2565(00)70605-9
21. The combinations of polymorphisms in vitamin D receptor, osteoprotegerin and tumour necrosis factor superfamily member 11 genes are associated with bone mineral density / S. Mencej-Bedrač [et al.] // J. Mol. Endocrinol. - 2009. - Vol. 42, N 3. - P. 239-247. https://doi.org/10.1677/jme-08-0108
22. Vitamin D receptor gene polymorphisms, bone mineral density and bone turnover: FokI genotype is related to postmenopausal bone mass / K. Zajicková [et al.] // Physiol. Res. - 2002. - Vol. 51, N 5. - P. 501-509.
23. Associations between VDR gene polymorphisms and osteoporosis risk and bone mineral density in postmenopausal women: a systematic review and meta-analysis / L. Zhang [et al.] // Sci. Rep. - 2018. - Vol. 8, N 1. - P. 981. https://doi.org/10.1038/s41598-017-18670-7
24. The polymorphism in the caudal-related homeodomain protein Cdx-2 binding element in the human vitamin D receptor gene / H. Arai [et al.] // J. Bone Miner. Res. - 2001. - Vol. 16, N 7. - P. 1256-1264. https://doi.org/10.1359/jbmr.2001.16.7.1256
25. Reference SNP (rs) Report: rs11568820 // National Center for Biotechnology Information [Electronic resource]. - Mode of access : https://www.ncbi.nlm.nih.gov/projects/SNP/snp_ref.cgi?rs=11568820. - Date of access : 07.04.2019.
26. Shen, H. Vitamin D receptor gene and risk of fracture in postmenopausal women: a meta-analysis / H. Shen, J. Xie, H. Lu // Climacteric. - 2014. - Vol. 17, N 4. - P. 319-324. https://doi.org/10.3109/13697137.2013.856401
27. Vitamin D receptor gene BsmI and TaqI polymorphisms and fracture risk: a meta-analysis / Y. Fang [et al.] // Bone. - 2006. - Vol. 39, N 4. - P. 938-945. https://doi.org/10.1016/j.bone.2006.04.016